Experimental Physiology
	

Celebrating 100 years
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH
 QUICK SEARCH:   [advanced]


     


Physiology in Press

First published online on September 21, 2006.
Experimental Physiology (2006)
DOI: 10.1113/expphysiol.2006.035493
© The Physiological Society 2006

A more recent version of this article appeared on November 1, 2006
This Article
Right arrow Full Text (Rapid PDF)
Right arrow All Versions of this Article:
91/6/943    most recent
expphysiol.2006.035493v1
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Casadei, B.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Casadei, B.

Received August 18, 2006
Revised September 12, 2006
Accepted after revision September 19, 2006


Cardiovascular Control [210]

The Joan Mott Lecture The emerging role of nNOS in the regulation of myocardial function

Barbara Casadei 1*

1 University of Oxford, John Radcliffe Hospital

* To whom correspondence should be addressed. E-mail: barbara.casadei{at}cardiov.ox.ac.uk.


   Abstract
The emerging role of nNOS in the regulation of myocardial function

Key Words: Calcium, Cardiac muscle, Nitric oxide




This article has been cited by other articles:


Home page
Int J EpidemiolHome page
M. D Tobin, M. Kahonen, P. Braund, T. Nieminen, C. Hajat, M. Tomaszewski, J. Viik, R. Lehtinen, G A. Ng, P. W Macfarlane, et al.
Gender and effects of a common genetic variant in the NOS1 regulator NOS1AP on cardiac repolarization in 3761 individuals from two independent populations
Int. J. Epidemiol., October 1, 2008; 37(5): 1132 - 1141.
[Abstract] [Full Text] [PDF]


Home page
Circ. Res.Home page
Y. H. Zhang, M. H. Zhang, C. E. Sears, K. Emanuel, C. Redwood, A. El-Armouche, E. G. Kranias, and B. Casadei
Reduced Phospholamban Phosphorylation Is Associated With Impaired Relaxation in Left Ventricular Myocytes From Neuronal NO Synthase-Deficient Mice
Circ. Res., February 1, 2008; 102(2): 242 - 249.
[Abstract] [Full Text] [PDF]


Home page
Proc. Natl. Acad. Sci. USAHome page
D. R. Gonzalez, F. Beigi, A. V. Treuer, and J. M. Hare
Deficient ryanodine receptor S-nitrosylation increases sarcoplasmic reticulum calcium leak and arrhythmogenesis in cardiomyocytes
PNAS, December 18, 2007; 104(51): 20612 - 20617.
[Abstract] [Full Text] [PDF]


Home page
Am. J. Physiol. Heart Circ. Physiol.Home page
X. Loyer, P. Oliviero, T. Damy, E. Robidel, F. Marotte, C. Heymes, and J.-L. Samuel
Effects of sex differences on constitutive nitric oxide synthase expression and activity in response to pressure overload in rats
Am J Physiol Heart Circ Physiol, November 1, 2007; 293(5): H2650 - H2658.
[Abstract] [Full Text] [PDF]


Home page
EndocrinologyHome page
X. Loyer, T. Damy, Z. Chvojkova, E. Robidel, F. Marotte, P. Oliviero, C. Heymes, and J.-L. Samuel
17{beta}-Estradiol Regulates Constitutive Nitric Oxide Synthase Expression Differentially in the Myocardium in Response to Pressure Overload
Endocrinology, October 1, 2007; 148(10): 4579 - 4584.
[Abstract] [Full Text] [PDF]




HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH
Copyright © 2006 by the The Physiological Society.